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CALL FOR ABSTRACTS
2009 UHMS ANNUAL SCIENTIFIC MEETING
Thursday 25 June – Saturday 27 June, 2009
Crowne Plaza Los Cabos, Mexico
ONLINE SUBMISSION
(Contact UHMS if you can not submit online)
DEADLINE FOR SUBMISSION: FEBRUARY 27, 2009
On behalf of the Undersea Hyperbaric Medical Society, the UHMS Program Committee is pleased to extend its call for abstract submission to the 2009 Annual Scientific Meeting. This year’s meeting will be held June 25-27, 2009 at the Crowne Plaza Los Cabos in Cabo, Mexico. For your convenience, the formatting and submission guidelines have been reprinted in the below section entitled “Abstract Formatting Guidelines.” Abstracts should be submitted via the UHMS website at www.uhms.org. For those unable to access the UHMS website or having difficulty with the online submission format, additional assistance with the submission process can be obtained by contacting the UHMS home office. Authors should ensure that all submission requirements are met, as incomplete submissions may be returned for modification or declined. The deadline for regular submission is FEBRUARY 27, 2009. Acknowledgement of receipt will normally be provided within one week. Notification regarding abstract final acceptance will be provided via e-mail no later than April 10, 2009. Please note that all abstracts accepted for presentation at the UHMS Annual Scientific Meeting will be presented in a poster format. At the discretion of the UHMS Program Committee, presenters may also be invited to present their abstract orally in the general meeting, resident’s/trainee competition (if eligible) or the Associates session (if eligible). Notification regarding selection for oral presentation will also be provided no later than April 10, 2009.
ABSTRACT USE PERMISSIONS AND RIGHTS:
Submission of abstract, poster and/or oral presentation materials for consideration for presentation at the Annual Scientific Meeting constitutes consent by the author(s) for the UHMS to use the materials submitted in whole or part as it sees fit. All abstracts, posters (including required PowerPoint/PDF submissions) and oral presentations (including PowerPoint slides) accepted by the UHMS Program Committee for presentation at the Annual Scientific Meeting may be subsequently used and/ or published by the Society in print or various electronic media at the discretion of the UHMS. Once accepted for presentation by the UHMS Program Committee, no abstract, poster or oral presentation may be withdrawn or excluded from being subject to this agreement.
SUBMISSION DEADLINES, EXTENSIONS AND CHANGES:
- DEADLINE FOR REGULAR SUBMISSION: FEBRUARY 27, 2009
- LATE SUBMISSION DEADLINE: No new submissions will be accepted after MARCH 27, 2009. All submissions received after the February 27, 2009 regular submission deadline will incur a $75.00 late submission fee. This fee will be invoiced upon abstract acceptance by the UHMS Program Committee and payment must be received by the UHMS home office prior to April 10, 2009 for the presentation to be included in the meeting.
- CHANGES TO AN ABSTRACT: If you require any changes to your abstract after notification of UHMS Program Committee acceptance, you will be charged a fee of $50.00. Payment of this fee must be received by the UHMS home office prior to April 10, 2009 for the changes to be implemented. Note that the abstract change fee will be waived if UHMS Program Committee initiated the request for changes.
- NO CHANGES to an abstract will be accepted after April 10, 2009.
ABSTRACT FORMATTING GUIDELINES:
Language – All abstracts are to be written in English. The Program Committee reserves the right to alter abstracts where the English structure makes comprehension difficult.
Abstract Body Format
- Must be 300 words or less, exclusive of title, author(s) names, and institutional affiliation(s).
- All submissions should be in Microsoft Word format (you can attach a Word file to your online submission)
- Only text and text tables will be accepted. NO graphs or images may be submitted.
- Contents should include the following sections:
- Introduction / Background
- Materials and Methods
- Results
- Summary / Conclusions
Suggested Presentation Category
Specify one of the following categories that best characterizes your presentation:
- Diving/Decompression Illness: Theory & Mechanisms
- Carbon Monoxide Poisoning
- Chamber Safety and Patient Management
- Wound Healing and Important Adjuncts
- HBO2 Therapy and Mechanisms
- Diving/Decompression Illness: Clinical
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SAMPLE ABSTRACT 1
CHARACTERISTICS OF THE HEADACHE ASSOCIATED WITH ACUTE CARBON MONOXIDE POISONING
Hampson NB , Hampson LA. Hyperbaric Medicine Department, Virginia Mason Medical Center , Seattle , Washington .
INTRODUCTION: Headache is the most commonly reported symptom in acute carbon monoxide (CO) poisoning. It is often described as throbbing and diffuse, however, a systematic characterization of CO-associated headache has never been published. This study examines the characteristics of CO-associated headache to determine whether any typical pattern exists which might prove useful in the diagnosis of CO exposure.
MATERIALS AND METHODS: Patients referred for hyperbaric oxygen (HBO2) treatment of acute CO poisoning were asked whether headache was part of their symptom complex. When present, specific details about the nature of the headache were collected from 100 consecutive patients through use of a standardized questionnaire.
RESULTS: Information on acute CO-associated headache was collected from 34 female and 66 male patients with mean carboxyhemoglobin (COHb) 21.3+9.3%. The most common sources of CO were motor vehicles (31), forklifts (23), or furnaces (11). Poisonings were accidental in 81%. The most common location for pain was frontal (66%), although more than one location was involved in 58%. Nature of the pain at any time during its course was dull in 72% and sharp in 36%. Headache was throbbing in 41%. Pain was continuous in 74% and intermittent in 16%. Peak intensity of pain did not correlate with COHb level. Symptoms commonly associated with headache included dizziness, lightheadedness, weakness, and nausea. Headache improved prior to HBO2 treatment in 72%, resolving entirely in 21%. Of those with residual headache, pain improved with HBO2 in 97%, resolving entirely in 44%.
CONCLUSIONS: The headache accompanying acute CO poisoning is extremely variable in nature. "Classic" throbbing diffuse headaches were rarely described by patients. There are no patterns which can be considered characteristic to aid in the diagnosis of CO-induced headache. Similarly, no trait was identified which might allow elimination of CO poisoning from the differential diagnosis of headache.
Suggested category: HBO Clinical
Authorizations: a) Y b) Y
Financial disclosure: a) N b) N c) nil d) N
CV: brief
SAMPLE ABSTRACT 2
SERUM S-100b AS A MARKER OF NEUROLOGICAL EVENTS IN GOATS FOLLOWING DIRECT DECOMPRESSION IN A SIMULATED DISABLED SUBMARINE SCENARIO
Jurd KM, Parmar K*, Seddon FM, Loveman GAM, Blogg SL, Thacker JC, Stansfield MRD , White MG, Hunt BJ*. Protection and Performance Dept, DERA Alverstoke, Gosport , UK . * Dept Haematology, Guys and St Thomas ' Hospital Trust, London , UK .
BACKGROUND: S100b is a glial protein used as a marker of cerebral damage in a number of clinical situations. Neurological decompression illness ( DCI ) is a major risk following rapid decompression to the surface in a disabled submarine (DISSUB) scenario if recompression facilities are unavailable. The aim of this study was to investigate levels of S-100b in goats under simulated DISSUB conditions where neurological DCI may occur.
METHODS: Adult female or castrated male goats (35-75 kg) underwent dry chamber air saturation between 55 and 85 fsw for 24 h followed by rapid decompression at 0.9 bar/min (n=37). Gross neurological involvement was determined by clinical assessment and post mortem examination. Venous blood samples were taken pre-dive and at 5, 30, 60, 90, 120, 180 and 240 mins post surfacing. S-100b was measured in serial samples by immunoluminometric assay.
RESULTS: Neurological DCI was mainly evident in the goats which had undergone the deeper profiles. A greater percentage of animals undergoing these deeper profiles also had increased levels of S-100b compared to pre-dive values. The percentage of animals at each depth with raised S100b increased from 0% at 55 fsw to 100% at 85 fsw. Increases in levels of S100b were evident at 5 or 30 mins in most cases, with levels continuing to rise for at least 120 mins.
CONCLUSION: This study demonstrates that S100b is raised in goats following "rapid decompression" from air saturation at depths below 55 fsw. S100b may be useful as a marker of cerebral events in neurological DCI .
BritishCrown Copyright 2001
Published with the permission of DERA on behalf of the controller HMSO
Suggested category: Decompression Illness
Authorizations: a) Y b) Y
Financial disclosure*: a) Y, b) Y, Manufacturer ILA Co. (Immunoluminometric Assay). Lead author is owner of
company. NOTE: *fictitious, created as an example only , c) Y, mm/dd/yyyy
CV: brief
DEADLINE FOR SUBMISSION: FEBRUARY 27, 2009
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